"There is no such thing as consensus science."
WHAT DO I NEED TO KNOW?
A basic background in biology and chemistry will provide the background necessary to understand the disease mechanism of post sepsis. What are spirochetes? What is OspA? Are there other models of fungal immunosuppression? What are the biomarkers of these diseases? What has the NIH said about all of this?
Read the complete charge sheets for the fullest understanding of "Lyme disease" and all the crazy antics by "scientists" to pretend that it's "just a knee." Use PubMed to search the literature for the latest on immunosuppression diseases, tolerance, cross tolerance, immune paralysis, OspA/Pam3Cys and other key terms that you will encounter on this site.
Most people refer to spirochetes as "bacteria." But they're not typical bacteria, which are generally managed by Toll-like Receptor 4 ("TLR4") in the immune system. These buggers are far worse.
Outer surface protein A, or OspA (pronounced "ah-spay") is a toxic, fungal-type, triacylated lipoprotein that is used to induce experimental sepsis. So how did the crooks get away with calling it a "vaccine?" The world would like to know.